Google Search Detects the Status of Your Upcoming Flights, Sends You Reminder Notifications

Google has had detailed flight information for awhile, but its new integration with Gmail means its flight information has become much more personalized. Just type my flights into Google and you'll see all your upcoming flights with detailed information.

Google hinted at this when it first launched its Gmail integration, and it looks like it's actually been active for awhile, but if you didn't know about it, it's a good tip. Google will even send you notifications when you have an upcoming trip in the upper right-corner of the window. I couldn't test this myself, since I don't have any upcoming flight confirmations in my email, but you can see a screenshot of the feature above from Google Operating System.

It still isn't quite as detailed as something like TripIt, but it's insanely quick and available right from Google, which we like a lot. Hit the link to read more.

Google Shows Flight Notifications | Google Operating System

Source: http://feeds.gawker.com/~r/lifehacker/full/~3/KWZ2GZxPEOo/google-search-detects-the-status-of-your-upcoming-flights-sends-you-reminder-notifications

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Personal Bankruptcy Tips To Help You File Like A Pro | Siben ...

TIP! An experienced bankruptcy attorney can guide you through the process successfully. There are a lot of lawyers that you can pick from.

Those dealing with personal bankruptcy filing are known to experience feelings of embarrassment, irritation and shame. Many live with the constant fear of being unable to repay their debts while also maintaining a decent standard of living. Even if you file for bankruptcy you still have options available to you. Read on to learn more.

TIP! If you are thinking about bankruptcy, stop charging your credit cards and borrowing money. Taking on a huge amount of debt will make it look as if you?re just going on a huge spending spree before attempting to have the slate wiped clean.

Filing for bankruptcy isn?t something to be ashamed of. It is common for people to feel as if filing for bankruptcy is a symbol of failure. This can cause depression. If you have researched all your options and logically came to the conclusion that this works best for you, view it as making a positive step in reclaiming your finances rather than seeing it as a failure.

Chapter 7

TIP! Before you proceed with your personal bankruptcy case, review your decisions to be certain that the choice you are making is the right. Look into credit counseling to see if it could help you work out of your debt without bankruptcy.

Before you make the decision to file Chapter 7 personal bankruptcy, take time to think about anyone it could affect. You can relieve yourself of any liability for debts that you may share with someone else through a Chapter 7 filing. However, your creditors will be able demand that your co-debtor pays the debt off in full.

TIP! In order to properly assist you with a bankruptcy filing, your lawyer needs to know about every significant debt that you currently have. This will mean providing a great deal of sensitive information: credit card companies, lenders, hospitals, personal debts and so forth.

It is wise to reconsider filing for divorce if your financial situation is grim. Many people find themselves filing for bankruptcy after a divorce. It is never foolish to think twice about seeking a divorce.

TIP! Consolidate a list of what you owe. Once you have an idea of who you owe and how much you owe, you can figure out if bankruptcy is really an option for you or not.

Meet with a few attorneys who offer free consultations before hiring one. Be certain to speak with an attorney, not their paralegal or law clerk, since they cannot give legal advice. Comparing different lawyers makes it possible to find one with whom you work well.

Free Consultations

TIP! Because bankruptcy is such a challenging time that a great deal of stress, both mental and emotional, may be involved. Engage a competent lawyer in order to avoid excess stress and keep everything on track.

Find a bankruptcy attorney who offers free consultations, and ask lots of questions. Since most attorneys offer free consultations, meet with a few attorneys before deciding who to hire. Make your decision after all of your questions have been answered. After the consultation, you are not immediately required to come up with a decision. After your consultations, do some additional research on each attorney you consider qualified for the job.

TIP! A personal bankruptcy settlement does not mean your debts are 100 percent discharged every time. If you file using chapter 13, this will allow you to reconstruct the debts that you have in order for creditors to get a part of the money you owe them.

Before you file for personal bankruptcy, weigh all of your options. There are many other options including debt consolidation and making payment plans with your creditors. If a foreclosure is your reason for filing look into your options with your bank first, such as a loan modification. Some lenders will make concessions rather than losing the money owed to bankruptcy. These concessions include waiving late fees, lowering interest rates, and changing the loan term. Most creditors will be willing to work out an option to avoid not getting paid at all.

TIP! Some attorneys offer a phone service for free where creditors will be referred to when they wish to contact you regarding an account that?s delinquent. You just provide the number, and they call to confirm that the debt is part of your bankruptcy.

The best bankruptcy lawyers will have a solid reputation. These are the lawyers you want to seek out. When you?ve found one, ask whether they provide free consultations. If they do, get all your financial statements ready and go see them. Once you arrive your attorney can advise you on the proper steps to take to file for bankruptcy.

TIP! Make sure that you disclose every bit of financial information on your bankruptcy petition. If you leave off even one tiny detail, you may end up in some serious trouble, but at the least your claim will be denied.

By now, you should be able to see that personal bankruptcy does not leave you doomed. It may be difficult at first, but you can overcome bankruptcy. Keep the advice here in mind so that you can work toward getting back on your feet

Source: http://www.sibensiben.com/long-island-injury-lawyer-blog/uncategorized/personal-bankruptcy-tips-to-help-you-file-like-a-pro-2-2-2-2

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Sandy and the Age of Superstorms (Little green footballs)

Share With Friends: | | Politics - Top Stories Stories, News Feeds and News via Feedzilla.

Source: http://news.feedzilla.com/en_us/stories/politics/top-stories/267365399?client_source=feed&format=rss

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ScienceDaily: Gene News

http://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usTue, 27 Nov 2012 18:53:48 ESTTue, 27 Nov 2012 18:53:48 EST60http://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.http://www.sciencedaily.com/releases/2012/11/121127130256.htm Some infants are more susceptible to potentially life-threatening breathing problems after birth, and rare, inherited DNA differences may explain why, according to new research.Tue, 27 Nov 2012 13:02:02 ESThttp://www.sciencedaily.com/releases/2012/11/121127130256.htmhttp://www.sciencedaily.com/releases/2012/11/121127101534.htm Scientists have broadened our understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.Tue, 27 Nov 2012 10:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121127101534.htmhttp://www.sciencedaily.com/releases/2012/11/121127094311.htm Researchers have discovered an alternative mechanism for activating rhe oncogene Ras that does not require mutation or hormonal stimulus.Tue, 27 Nov 2012 09:43:43 ESThttp://www.sciencedaily.com/releases/2012/11/121127094311.htmhttp://www.sciencedaily.com/releases/2012/11/121127094248.htm Scientists have discovered that injecting a novel human protein into muscle affected by Duchenne muscular dystrophy significantly increases its size and strength, findings that could lead to a therapy akin to the use of insulin by diabetics.Tue, 27 Nov 2012 09:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121127094248.htmhttp://www.sciencedaily.com/releases/2012/11/121127093951.htm Researchers have identified and ?switched off? a chemical chain that causes neurodegenerative diseases such as Huntington?s disease, amyotrophic lateral sclerosis and dementia. The findings could one day be of particular therapeutic benefit to Huntington?s disease patients.Tue, 27 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121127093951.htmhttp://www.sciencedaily.com/releases/2012/11/121126164003.htm PKM2 slips into nucleus to promote cancer; potential biomarker and drug approach discovered.Mon, 26 Nov 2012 16:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121126164003.htmhttp://www.sciencedaily.com/releases/2012/11/121126142855.htm Discovery of a new drug with high potential to treat Ewing sarcoma, an often deadly cancer of children and young adults, and the previously unknown mechanism behind it, come hand-in-hand in a new study.Mon, 26 Nov 2012 14:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121126142855.htmhttp://www.sciencedaily.com/releases/2012/11/121126131349.htm A newly discovered gene that is essential for embryo survival could also hold the key to treating and potentially controlling chronic infections such as HIV, hepatitis and tuberculosis. The gene, called Arih2, is fundamental to the function of the immune system -- making critical decisions about whether to switch on the immune response to an infection.Mon, 26 Nov 2012 13:13:13 ESThttp://www.sciencedaily.com/releases/2012/11/121126131349.htmhttp://www.sciencedaily.com/releases/2012/11/121126110737.htm Two years ago, a 71-year-old Indiana man impaled his hand on a branch after cutting down a dead tree. The wound caused an infection that led scientists to discover a new bacterium and solve a mystery about how bacteria came to live inside insects.Mon, 26 Nov 2012 11:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121126110737.htmhttp://www.sciencedaily.com/releases/2012/11/121125192838.htm Over a decade after sequencing the human genome, it has now become clear that the genome is not mostly ?junk? as previously thought. In fact, the ENCODE project consortium of dozens of labs and petabytes of data have determined that these ?noncoding? regions house everything from disease trait loci to important regulatory signals, all the way through to new types of RNA-based genes.Sun, 25 Nov 2012 19:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121125192838.htmhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmhttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmhttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmhttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmhttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmhttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmhttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmhttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmhttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmhttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmhttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htmhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmhttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmhttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmhttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htmhttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmhttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmhttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmhttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmhttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmhttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmhttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmhttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmhttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmhttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htmhttp://www.sciencedaily.com/releases/2012/11/121115132613.htm New research has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy.Thu, 15 Nov 2012 13:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115132613.htmhttp://www.sciencedaily.com/releases/2012/11/121115132344.htm Thanks to some careful detective work, scientist better understand just how iPS cells form ? and why the Yamanaka process is inefficient, an important step to work out for regenerative medicine. The findings uncover cellular impediments to iPS cell development that, if overcome, could dramatically improve the efficiency and speed of iPS cell generation.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132344.htmhttp://www.sciencedaily.com/releases/2012/11/121115132342.htm About 10 percent of kids born with kidney defects have large alterations in their genomes known to be linked with neurodevelopmental delay and mental illness, a new study has shown.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132342.htmhttp://www.sciencedaily.com/releases/2012/11/121114172833.htm Relatively small levels of exposure to alcohol while in the womb can influence a child's IQ, according to a new study using data from over 4,000 mothers and their children.Wed, 14 Nov 2012 17:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121114172833.htmhttp://www.sciencedaily.com/releases/2012/11/121114171710.htm A gene so powerful it nearly triples the risk of Alzheimer's disease has been discovered by an international team of researchers. It is the most potent genetic risk factor for Alzheimer's identified in the past 20 years.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171710.htmhttp://www.sciencedaily.com/releases/2012/11/121114171708.htm For the first time, researchers have landed on a potential diagnostic method to identify at least a subset of patients with chronic fatigue syndrome - testing for antibodies linked to latent Epstein-Barr virus reactivation.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171708.htmhttp://www.sciencedaily.com/releases/2012/11/121114153227.htm In laboratory experiments, scientists have eliminated metastasis, the spread of cancer from the original tumor to other parts of the body, in melanoma by inhibiting a protein known as melanoma differentiation associated gene-9 (mda-9)/syntenin.Wed, 14 Nov 2012 15:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114153227.htmhttp://www.sciencedaily.com/releases/2012/11/121114134512.htm Researchers provide a whole-genome sequence and analysis of number of pig breeds, including a miniature pig that serves a model for human medical studies and therapeutic drug testing.Wed, 14 Nov 2012 13:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121114134512.htm

Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

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Powerball jackpot climbs to $550M on ticket sales

Pat Powell, 30, of Atlanta, buys a Powerball lottery ticket at a convenience store, Wednesday, Nov. 28, 2012, in Atlanta. "I think my odds are zero to zero," says Powell "I don't think I'm going to win but I'll just join the hype. If I did win, I'd open up my own business, an internet caf? in the West Indies and have a learning center here in Georgia. I'll invest and try to be as smart with it as I can with it. I will say for the past 3 days, for whatever reason, I've been thinking about winning this money and what I'd do with it. There's no ritual but it's just been on my mind so it's like, let me just join the hype and just do it." (AP Photo/David Goldman)

Pat Powell, 30, of Atlanta, buys a Powerball lottery ticket at a convenience store, Wednesday, Nov. 28, 2012, in Atlanta. "I think my odds are zero to zero," says Powell "I don't think I'm going to win but I'll just join the hype. If I did win, I'd open up my own business, an internet caf? in the West Indies and have a learning center here in Georgia. I'll invest and try to be as smart with it as I can with it. I will say for the past 3 days, for whatever reason, I've been thinking about winning this money and what I'd do with it. There's no ritual but it's just been on my mind so it's like, let me just join the hype and just do it." (AP Photo/David Goldman)

Lamar Fallie, 52, of Chicago buys six Powerball tickets at a BP gas station Wednesday, Nov. 28, 2012 in Calumet Park, Ill. Fallie who is currently unemployed say he doesn't normally play the lottery but was lured by tonight's 500 million dollar jackpot. If he wins he says he will take care of his church first, then every child Beasley Elementary School will get a laptop, he will make a donation to Julian High School then he will retire from being unemployed. (AP Photo/M. Spencer Green)

Norn Phneo sells Powerball tickets to Billy Fulginiti, of Philadelphia for himself and his coworkers, Wednesday, Nov. 28, 2012, in Philadelphia. (AP Photo/Matt Rourke)

Canterbury Country Store manager Toni Halla smiles as she rings in another powerball customer, Wednesday, Nov. 28, 2012 in Canterbury, N.H. Halla says sales in the small town of 12,000 people has been brisk. (AP Photo/Jim Cole)

Retired Dr. Paul Kruzel kisses his lottery ticket for luck after buying it at the Canterbury Country Store Wednesday, Nov. 28, 2012 in Canterbury, N.H. (AP Photo/Jim Cole)

CHICAGO (AP) ? As Americans went on a ticket-buying spree, the Powerball jackpot rose to $550 million Wednesday, enticing many people who rarely, if ever, play the lottery to purchase a shot at the second-largest payout in U.S. history.

Among them was Lamar Fallie, a jobless Chicago man who said his six tickets conjured a pleasant daydream: If he wins, he plans to take care of his church, make big donations to schools and then "retire from being unemployed."

Tickets were selling at a rate of 130,000 a minute nationwide ? about six times the volume from a week ago. That meant the jackpot could climb even higher before the Wednesday night drawing, said Chuck Strutt, executive director of the Multi-State Lottery Association.

The jackpot has already rolled over 16 consecutive times without a winner, but Powerball officials say they now believe there is a 75 percent chance the winning combination will be drawn this time.

If one ticket hits the right numbers, chances are good that multiple ones will, according to some experts. That happened in the Mega Millions drawing in March, when three ticket buyers shared a $656 million jackpot, which remains the largest lottery payout of all time.

Yvette Gavin, who sold the tickets to Fallie, is only an occasional lottery player herself, but the huge jackpot means she'll definitely play this time. As for the promises she often gets from ticket purchasers, Gavin isn't holding her breath.

"A lot of customers say if they win they will take care of me, but I will have to wait and see," she said.

In the hours before Wednesday's drawing, Associated Press photographers across the nation sought out ticket buyers and asked about their lottery fantasies. Here's a look at what they found:

___

When Atlanta barber Andre Williams buys scratch-off tickets, he typically does a dance in his shop for good luck. As a first-time Powerball player, he plans to reprise the dance ? and buy a few extra tickets to enhance his chances.

I don't even know if I'll look at it," said Williams, who bought his ticket at a newsstand. "If I win, I might pass out."

Paralegal Pat Powell was buying her first Powerball ticket at another store in Atlanta, even though she acknowledged her odds were probably "zero to zero."

Still, Powell has specific plans should she win: start an Internet cafe in the West Indies and a learning center in Georgia.

"I've been thinking about winning this money and what I'd do with it," Powell said. "There's no ritual, but it's just been on my mind. So it's like, let me just join the hype and just do it."

Atlanta accountant Benita Lewis, who had never played the lottery before, didn't want to be the only one left in her office without a ticket.

"I did feel nervous buying it like I could be the one," she said. "I'm going to retire and pay off all my family's debt."

___

In Philadelphia, seafood salesman Billy Fulginiti bought 50 Powerball tickets with co-workers and a few more with a small group. He said he only plays when the jackpot is especially large.

"You go to bed at night wishing you wake up a millionaire," Fulginiti said. He planned to take a long vacation and "help a lot of people, a lot of charities," if any of his tickets turn out to be winners.

___

Powerball purchases at the Canterbury Country Store in Canterbury, N.H., have been so steady that the manager has been working extra evening hours to keep up.

Horticulturist Kevin Brags buys tickets at the store two to three times a month. He says he usually picks numbers higher than 32 because so many people use numbers 31 and lower, largely because of birthdays.

The birthday theory didn't scare off Paul Kruzel, a retired doctor who chooses the days his children were born.

Both, however, have the same plans for winning: "make a lot of people happy."

John Olson has a more elaborate idea: He'd like to buy an island.

___

At a downtown Detroit convenience store, Ceejay Johnson purchased five Powerball tickets. If she strikes it rich, the analyst from Southfield, Mich., said she would buy a home for her sister in Florida. Then she would "go into hiding" and take care of her family.

"And the IRS," she added.

___

Associated Press photographer Jim Cole reported from Canterbury, N.H.

___

Associated Press photographers Paul Sancya in Detroit, David Goldman in Atlanta and Matt Rourke in Philadelphia, and AP writers David Pitt in Des Moines, Iowa, and Jeff McMurray in Chicago contributed to this report.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/386c25518f464186bf7a2ac026580ce7/Article_2012-11-28-Powerball-Jackpot/id-f26437641af54c0c9f6838d14d083226

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Video: PRESS Pass: Fmr. FDIC Chairman Sheila Bair

Sorry, Readability was unable to parse this page for content.

Source: http://www.msnbc.msn.com/id/3032608/vp/50000683#50000683

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Rev. Run Weight Loss: Hip-Hop Legend Drops 22 Pounds In Effort ...

At 48, Joseph "Rev. Run" Simmons is finally hitting his stride with a healthy diet and exercise routine that he says is on par with achieving the kind of physique he'll need to keep up with his Run-D.M.C. group mate Darryl "D.M.C" McDaniels.

"I just did a concert with D.M.C. after 13 years, and after losing some pounds for that, I felt really good," Simmons told The Huffington Post. "We got good reviews for having lots of energy on stage and I realized that if I?m going to continue to do shows ... I gotta be in Mick Jagger-type of condition."

But aesthetic goals (and criticism from the public) aside, Simmons says his new healthy-living routine is rooted in something far more serious -- combating his higher-than-average risk for Type 2 diabetes.

"I found out that [my father] had diabetes in his older age and as I was moving along in my life ... I realized that I had to start losing weight," he said, explaining how he's taken up walking and recently dusted off his treadmill in an effort to stay the course during the chillier winter months. "[It was] for many reasons, but the number one reason was for health."

And while family history factors in to why Simmons needs to get his health in check, diabetes educator Jeannette Jordan said it's a myth many people need to dispel.

"One of the biggest [misconceptions] out there is people feel like diabetes runs in [their] family, so it?s inevitable that they?re going to get it," Jordan said, touting her employer Novo Nordisk's "Ask. Screen. Know." campaign, for which Simmons is now an ambassador. "The message that we?re trying to get out is that it doesn?t have to happen to you the same way. Yes, family history is a risk factor, but it doesn?t mean that because this happened to your mom it has to happen to you.

A diabetes diagnosis also doesn't mean life-altering changes, Jordan said. "What we need to do is not that difficult ... moderation is actually the key. We don?t take away everything that you love, it?s just how much we eat," Jordan said, stressing the importance of knowing which foods turn into sugar, forgoing sweetened soft drinks, and realizing that you can limit yourself to just one cheddar biscuit from Red Lobster and still enjoy your meal.

"The myth is that life is going to change so drastically, '[I'm] not going to be happy with my dining anymore', and that?s not true. You can live well with diabetes if you have it. But the first thing is, you have to be screened, you have to know if you have it. Ask yourself if you?re at risk and then do something about that," Jordan said.

Simmons agrees that knowing is the key -- even if you are the picture of perfect health. "My dad had diabetes, so it doesn?t mean just because Russell stands on his head and does yoga, and Angela?s in perfect health that they don?t have it," he said, referring to his yoga-promoting brother Russell Simmons and his daughter Angela Simmons, who showed off a svelte new figure earlier this year.

And just ahead of the holidays Simmons said he's relying on them for support. "They?re right there for me; they know what I?m doing to keep my weight under control ... and [during the holiday season] I believe in being proactive instead of reactive," Simmons said. "I have all types of things set up so I know, this is what I?m going to eat when this sweet tooth moment comes, this is what I?m going to eat when dinner comes around. And no, I?m not going to wait until the big dinner. [For Thanksgiving], I didn?t wait for the one big moment, I had already eaten twice, the right way, before dinner, so when dinner came, I wasn?t starved."

Related on HuffPost:

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Source: http://www.huffingtonpost.com/2012/11/27/rev-run-weight-loss-lower-diabetes-risk_n_2198731.html

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PressNewsRoom ? Blog Archive ? Real Estate Expert Mark Torok ...

Mark Torok, Principal of The Torok Law Firm P.C., was recently seen on ABC and other network affiliates across the country as an expert guest on ?New Masters of Real Estate.?

San Antonio, TX. ? Mark Torok, Principal of The Torok Law Firm P.C., was recently an expert guest on ?New Masters of Real Estate.? Emmy Award Winning Director and Producer, Nick Nanton, hosts the show that was recently featured on ABC and other major network affiliates across the country.

?New Masters of Real Estate? features an interview format, with Nick Nanton interviewing the nation?s leading real estate agents. Mark was one of Nick?s recent guests, discussing his success and knowledge in real estate, and in particular, subject-to real estate transactions.

Mark Torok founded The Torok Law Firm P.C. to represent real estate investors for real estate, asset protection, securities and estate planning. Guiding investors through the world of real estate investing with classes and training products covering the legal side of real estate investments is the driving purpose of Torok Law Education.
Mark has closed more than 500 transactions and represents some of the largest investors in Texas, as well as several hard money?lenders. He is an active real estate investor and currently owns more than 800 units in and around Texas. He holds active Texas and Pennsylvania?admissions?to practice law, a real estate salesperson?s license, an escrow agents license and works with Providence Title to provide superior title services to his clients.

Mark has had a varied and extensive legal career of almost 30 years, including stints as an administrative law judge in Oregon and Pennsylvania, representing a national insurance trade association for legislative and regulatory affairs for six states, where he was instrumental in the design and passage of major legislation, and as an officer and counsel to three major insurance companies.

To learn more about Mark Torok, please visit www.toroklaw.com

Source: http://www.pressnewsroom.com/index.php/2012/11/28/real-estate-expert-mark-torok-featured-on-abc-as-guest-on-%E2%80%9Cnew-masters-of-real-estate%E2%80%9D/

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The Ugly Duckling House | DIY Home Improvement Blog: Tips for ...

Baseboards may not be the most expensive thing to buy in the molding department, but if you're doing a renovation and are able to save the pieces you remove from the wall intact, your savings can really add up. ?Here are just a few of my tips for salvaging old baseboards:

Removing baseboards

*note: the steps in this section were done while I was in class (which is nicer than coming home after class and still having to do it - thanks Dad!), so I don't have pictures to show. The next time I have to remove them, I'll take these and update the post.

1. If it is difficult to distinguish where the wall ends and the baseboard begins, this usually means that there is caulk in the seam. ?This usually makes things look better than leaving a small gap where the wall and baseboard meet. ?In my case, there was no caulk, so this step was skipped. ?If there is, cut the old caulk where the baseboard meets the wall. ?Be sure to slice downward (between the wall and board) so you don't cut into your drywall. ?This preps the baseboard so that it will more easily separate from the wall in the next step.

2. Use a flat prybar to wedge between the baseboard and wall. ?I own this set and recommend it.?(affiliate link)? Use the large, flat one. ?Try to be gentle, but pry the baseboard away. ?If you can simply remove the baseboard, nails and all, do it. ?Pretty Handy Girl has an excellent visual example if you need one of this (since I don't have the pictures yet!). ?If you can't pry the board off cleanly without splintering the board, try to pry just enough off the wall, then hit the baseboard back onto the wall with a swift blow of a hammer (but not where the nails lie; you'll want space along the wall to create the effect). ?What often happens is the nail will stay more firmly in place, but the hammer's connection to unsupported (no nail) spots on the board will further separate the nails from the wood. ?Often this causes the nail to jut out from the baseboard at this point, so you can use a claw hammer to completely remove the nails and take the baseboard off.

Another baseboard nail removal method

As you can see from the picture below, the original nail holes were never covered when the baseboard was installed, so nail removal was a snap. ?If you pried the baseboard off the wall complete with nails poking out of the board, try the following technique: 1. Flip the board over to expose the nails. ?I found that using a soft surface (like the carpet just before my upstairs hallway - excuse the debris from working in the guest bath!) worked easier since it was more of a cushion.
2. Hit the nail points with a hammer, causing the nails to shift back through the front of the board.
3. Flip the board over again. ?With the claw part of the hammer, pry the rest of the nail out of the wood. ?(If you have a completely unblemished front and do not wish to pull the nail through the front, you can use pliers to pull the nail through on the back side, but I've found this takes a little more muscle sometimes so I'd rather use the front).

Painting baseboards

I highly recommend priming and applying at least the first coat of paint to the baseboards when it is still off the wall. ?It is simply faster and you don't have to tape anything off. ?The pic below is after two coats. ?Don't they look a million times better now?

Re-installation of baseboard trim

The best part about re-using the baseboards is that your cuts are already made (unless you make changes to the width of something that was installed).

1. (Optional - and useful where there are areas that can't be nailed in place, like around my tub). ?Apply construction adhesive to the back of the baseboard in an s-curve fashion. ?Using circular motions will cause air bubbles.

2. The old nail holes might be too weak to use again, so I would recommend finding a new spot to drive a new nail (especially if you're painting it anyway). ?Just a couple nails every eight inches or so (if your molding is like mine, top and bottom in one spot) should do it. ?If you're working around hard surfaces like laminate or tile, be sure not to angle the nail's entry so that it doesn't bounce off the surface (and fly at your face; I've seen it happen). ?Regardless, be sure you use eye protection if you're using a nail gun.

3. If the baseboards do not fit (I had one spot where my new vanity was wider than the old, so installing the old baseboard was about an inch too long), use an oscillating tool or miter saw to cut off the excess. ?Again, this is much easier and cheaper than starting fresh, even if there are a few adjustments to make.

4. When everything is back in place, caulk the top (where the board meets the wall) to keep things clean (no one likes crevices, even tiny ones, where dust can settle) and caulk any seams or nail holes, then touch up with paint.

Prest-o, change-o, cheap-o. ?Use the dollars you saved to buy yourself another tool (or whatever else you like to spend your money on - that's just my choice).

Source: http://www.uglyducklinghouse.com/2012/11/tips-for-saving-and-reusing-old.html

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Mitel set to retire the 5310 Conference Unit | Denwa ...

Posted on 26 November 2012. Tags: mitel 5310, mitel 5310 discontinue, mitel 5310 retired, mitel conferencing unit

Mitel have recently issued a press release notifying resellers that the discontinuance Mitel 5310 will be effective as of the 1^st May 2013. Mitel have come to this decision because of the successful introduction of their UC360 Collaboration Point which offers more features such as rich audio and data sharing facility.

Mitel do intend to manufacture discontinue the 5310 Conference unit so supplies of the 5310 may not be available before the discontinue date of the 31^st May. Mitel will make every effort to sources any spare parts that may be required either new or refurbished for a period of 5 years or until all spare parts are depleted.

Although Mitel cannot guarantee delivery of the Mitel 5310 they will try to fulfill all purchase orders containing the 5310 up until the 31^st May 2013 so if you do wish to purchase one the time to buy would be now.

Source: http://blog.denwa.uk.com/mitel-set-to-retire-5310-conference-unit/

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LeadSift Raises $500K To Make Twitter, Foursquare More Fertile Ground For Sales Leads

Make way for another company that's helping enterprises make sense of social media. LeadSift, a Canada-based social media startup that helps businesses mine social data on sites like Twitter and Foursquare for sales lead generation, has raised a seed round of $500,000 from Omers Ventures, the fund that invests in other Canada-based social media startups like Hootsuite. The company says that it will use the funds to continue developing its product -- still in beta with a small group of customers -- marketing and staffing.

Source: http://feedproxy.google.com/~r/Techcrunch/~3/l3Xei6l4U9M/

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Cuomo likens Katrina, Sandy as damage total soars

ALBANY, N.Y. (AP) ? New York Gov. Andrew Cuomo declared Superstorm Sandy in some ways worse than 2005's Hurricane Katrina as he said his state would need $42 billion to recover from the damage wreaked in late October and prevent future catastrophe.

The figure includes more than $32 billion for damage and restoration and an additional $9 billion to head off damage in future storms, including steps to protect the power grid and cellphone network.

As he and other political leaders in his state conferred on how much federal aid to seek, he said New York taxpayers can't foot the bill.

"It would incapacitate the state," he said at a news conference Monday. "Tax increases are always a last, last, last resort."

Comparisons of Sandy to Katrina, which swamped New Orleans and the Gulf Coast in 2005, put the East Coast's recovery "in focus," he declared, saying Sandy hit a more densely populated region and caused more costly damage than Katrina.

Katrina killed more than 1,800 people, flooded nearly the entire city of New Orleans and caused some $108 billion in damage. Sandy killed more than 100 as it swamped coastal areas, toppled trees and dumped snow inland, and the most recent estimates indicate damage totaling more than $62 billion in several Eastern states, with New York and New Jersey accounting for the lion's share.

Previous estimates, which often fluctuated, had put Sandy's damage at around $50 billion. That already made it the second most destructive U.S. storm in history, after Katrina.

New Jersey Gov. Chris Christie, who announced Monday that he couldn't abandon the state during its recovery and would seek re-election next year, has put the preliminary damage estimate in his state at $29.5 billion.

"It would be wrong for me to leave now," said Christie, a Republican who controversially lauded President Barack Obama for his attentiveness after the storm. "I don't want to leave now. We have a job to do. That job won't be finished by next year."

States typically get 75 percent reimbursement for the cost of governments to restore mass transit and other services after a disaster. The Federal Emergency Management Agency has paid out nearly $248 million already in New Jersey.

In New York, Cuomo, a Democrat, met with his state's congressional delegation to discuss the new figures and present "less than a wish list." The delegation, Cuomo and New York City Mayor Michael Bloomberg will now draw up a request for federal disaster aid.

Bloomberg had announced earlier in the day that Sandy caused $19 billion in losses in New York City ? part of the $32 billion estimate Cuomo used.

Most basic recovery costs for roads, water systems, schools, parks, individual assistance and more total $15 billion in New York City; $7 billion for state agencies; $6.6 billion in Nassau County and $1.7 billion in Suffolk County, both on suburban Long Island; and $527 million in Westchester County and $143 million in Rockland County, both north of New York City, according to a state document used in the private briefing of the delegation and obtained by The Associated Press.

Hard times were already facing the state and city governments that were staring at deficits of more than $1 billion before Sandy hit in late October. State tax receipts have also missed projections, showing a continued slow recovery from a recession that could hit taxpayers in the governments' budgets this spring. And there's the looming fiscal cliff, the combination of expiring federal tax cuts and major spending cuts that could rattle the economy.

"Make no mistake, this will not be an easy task, particularly given the impending fiscal cliff, and a Congress that has been much less friendly to disaster relief than in the past," said Sen. Charles Schumer, a powerful New York Democrat.

"We will work with the (Obama) administration on supplemental legislation, to be introduced in the upcoming December session of Congress, that will set us on the road to meeting New York's needs," he said. "This will be an effort that lasts not weeks, but many months, and we will not rest until the federal response meets New York's deep and extensive needs."

___

Contributing to this report were Associated Press writers Jennifer Peltz and Meghan Barr in New York City and Angela Delli Santi in Trenton, N.J.

Source: http://news.yahoo.com/cuomo-likens-katrina-sandy-damage-total-soars-072117574--finance.html

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10-year-old girl's laptop confiscated after copyright offense

Featured

11 hrs.

Helen A.S. Popkin

If you're confused over a recent email from Facebook regarding its data use policy, you're not alone.? The email ? with the subject line "Up... Read more

5 hrs.

Devin Coldewey , NBC News

Police in Helsinki?seized?the laptop of a young girl during a search of her family's home last week, according to her father.?The alleged?offense? Using the?popular BitTorrent website The Pirate Bay to download a single album.

Last year, 9-year-old Julietta?came across a torrent on The Pirate Bay after searching on Google?for Finnish pop star Chisu's latest album. The download failed to work, and she and her father went and bought the album together shortly afterwards.?Unbeknownst to them,?Finland's Copyright Information and?Anti-Piracy Centre (known as CIAPC, as well as its Finnish acronym, TTVK) had already?taken notice.

The events are related by the girl's father,?Aki Wequ Nylund,?in?a public?Facebook post. (Though Google Translate's Finnish is not very good, an account of the translated?story?was posted at copyright and BitTorrent news blog?TorrentFreak.)

That spring, a letter arrived from the TTVK?alleging that the Nylund's account had been linked to a copyright infringement. The letter gave the option to pay a settlement of ?600 and sign a non-disclosure agreement ? a common tactic used by copyright holders that removes the need for formal charges.

Nylund contacted the?TTVK?lawyer to contest the matter, but?the TTVK continued its pursuit of damages. Last Tuesday morning, he found a pair of Finnish police officers standing at his doorstep.

The police presented a search?warrant, entered, and identified the girl's Winnie?the?Pooh-decorated laptop as the object of their search, and confiscated it.

Unsurprisingly, the events have drawn criticism locally and abroad. Finnish Internet rights watchdog Electronic Frontier Finland denounced the actions of the TTVK in a statement and blog post, calling attention to the arbitrary nature of the settlements and their use?as a scare tactic. They also point out that?Nylund's acknowledgment?in this case notwithstanding, an IP address used to track an infringement cannot be linked to a person's identity.

The TTVK's executive director defended the actions in comments to Finnish newspaper Helsingin Sanomat, saying that TTVK and the police were only working to enforce the law.?

Chisu, the pop star whose album was at the center of the controversy, expressed in a Facebook post that she supported copyright law but apologized for the situation.

Niko?Nordstr?m, CEO of Warner Music Finland, acknowledged (also via Chisu's Facebook page)?the limits of IP-based enforcement, but said?"this procedure is currently the only way to tackle illegal downloading" (translation by Google).

Had the TTVK known that the infringing party in this situation was a young?girl, might they?have taken a softer approach? In past cases,?U.S. media associations have not?made accommodations for minors, either.

As for Julietta's lost laptop, an anonymous benefactor sent a brand-new MacBook Pro to replace it. Her father reports that she is already putting it to use, playing online with her friends. An administrator at The Pirate Bay promised VIP privileges to her as well if she wants them?? ?although after this experience, it would be understandable if she opted not to take advantage of the offer.

Devin Coldewey is a contributing writer for NBC?News Digital. His personal website is?coldewey.cc.

Source: http://www.nbcnews.com/technology/technolog/10-year-old-girls-laptop-confiscated-after-copyright-offense-1C7227561

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Kipling Seoul Laptop Large Backpack True Blue ? Travel & Leisure ...

The Seoul Large Backpack from Kipling has laptop protection and padded shoulder straps making it great for travel or school. Main zipped compartment contains laptop pocket with Velcro closure, and a padded back and bottom panel. Zip-front pocket contains 2 pen sleeves, cell phone pocket, iPod/PDA pocket and large internal zippered pocket. Additional large zippered front pocket for more storage. Padded straps that are adjustable to a maximum length of 32 inches and top grip handle. Zippered pockets at the top and left hand side of bag are perfect for quick access to small items.

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Source: http://boyajianmarc.com/travel/2012/11/19/kipling-seoul-laptop-large-backpack-true-blue/

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Seth's Blog ? Blog Archive ? Thirty Years of Breast Cancer ...

A recent op-ed in the New York Times by H. Gilbert Welch, a co-author of Overdiagnosis, describes a tragedy of ignorance and overconfidence. The current emphasis on regular mammograms began thirty years ago. They will prevent breast cancer, doctors and health experts told hundreds of millions of women. They will allow early detection of cancers that, if not caught early, would become life-threatening. The campaign was very successful. According to the paper cited by Welch, about 70% of American women report getting such screening.

It is now abundantly clear this was a mistake. If screening worked perfectly ? if all of the cancers it detected were dangerous ? the rate of late-stage breast cancer should have gone down by the amount that the rate of early-stage breast cancer went up. Over the thirty years of screening, the rate of (detected) early-stage breast cancers among women over 40 doubled, no doubt because of? screening. (Over the same period the rate of early-stage breast cancers among women under 40 barely changed.) In spite of all this early detection and treatment, the rate of late-stage breast cancer among women over 40 stayed essentially the same. All that screening (billions of mammograms), all that chemo and surgery and radiation, all that worry and time and misery ? and no clear benefit to the women screened and those who paid for the screening, treatment, and so on. Roughly all of the ?cancers? detected by screening and then, at great cost, removed, aren?t dangerous, it turns out.

Quite apart from the staggering size of the mistake and the long time needed to notice it, screening has been promoted with specious logic.

Proponents have used the most misleading screening statistic there is: survival rates. A recent Komen Foundation campaign typifies the approach: ?Early detection saves lives. The five-year survival rate for breast cancer when caught early is 98 percent. When it?s not? It decreases to 23 percent.? Survival rates always go up with early diagnosis: people who get a diagnosis earlier in life will live longer with their diagnosis, even if it doesn?t change their time of death by one iota.

Did those making the 98% vs. 23% argument not understand this?

I applaud Welch?s research, but his op-ed has gaps. A unbiased assessment of breast cancer screening would include not only the (lack of) benefits but also the (full) costs. Treatment for a harmless ?cancer? may cause worse health than no treatment. Maybe chemotherapy and radiation and surgery increase other cancers, for example. What about the effect of all those mammograms on overall cancer rate? Welch fails to consider this.

Welch also fails to make the most basic and important point of all. To reduce breast cancer, it would be a good idea to learn what environmental factors cause it. (For example, maybe poor sleep causes breast cancer.) Then it could be actually prevented. Much more cheaply and effectively.? Yet the Komen Foundation and the Canadian Breast Cancer Foundation say ?race for the cure? instead of trying to improve prevention.

?

This entry was posted on Sunday, November 25th, 2012 at 5:00 am and is filed under doctor logic, health care. You can follow any responses to this entry through the RSS 2.0 feed. You can leave a response, or trackback from your own site.

Source: http://blog.sethroberts.net/2012/11/25/after-thirty-years-still-plausible-that-breast-cancer-screening-does-more-harm-than-good/

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Gene Chizik Fired: Auburn Fires Football Coach After 3-9 Season

AUBURN, Ala. ? Gene Chizik led Auburn to a historic season but couldn't keep the Tigers from descending to their lowest point in decades.

The rapid fall from a national championship to 3-9 and the Southeastern Conference doormat led to Chizik's firing Sunday, the day after a humbling 49-0 loss to No. 2 Alabama that showed just how far the program has fallen.

The Tigers endured the worst slide within two years of winning a national championship of any team since the Associated Press poll started in 1936 and hadn't lost this many games since going 0-10 in 1950. The decision came 17 months after Auburn gave Chizik a contract worth some $3.5 million annually through 2015 with a hefty buyout.

Auburn athletic director Jay Jacobs said he informed Chizik Sunday morning after President Jay Gogue accepted his recommendation to fire the coach.

The players were informed in a team meeting Sunday. Jacobs scheduled a news conference for later in the afternoon.

"I'm extremely disappointed with the way this season turned out and I apologize to the Auburn family and our team for what they have had to endure," Chizik said in a statement released by Auburn. "In my 27 years of coaching, I have gained an understanding of the high expectations in this profession. When expectations are not met, I understand changes must be made."

The Tigers went from 14-0 with a perfect SEC record with Cam Newton leading the offense in 2010 to 3-9 and 0-8, losing their final three league games by a combined 150-21. Auburn was blown out by Texas A&M (63-21) and Georgia (38-0) but the finale was even more painful for Tigers fans.

The Crimson Tide cruised to a six-touchdown halftime lead and the second-most lopsided Iron Bowl victory in history, behind only the Tide's 55-0 win in 1948.

"While we experienced a tremendous low in 2012, I will always be proud of the incredible highs that we achieved, including three bowl victories, an SEC championship and a national championship," Chizik said.

He was 33-19 in four seasons and 15-17 in SEC games.

Auburn said the total buyout for Chizik and his assistant coaches is $11.09 million. Chizik's buyout is expected to total $7.5 million and will be paid in monthly installments for the next four years.

Six assistants are under contract through June 30, 2013 while defensive coordinator Brian VanGorder, offensive coordinator Scot Loeffler and wide receivers coach Trooper Taylor have deals extending another year beyond that.

The buyouts could be reduced if the coaches find other jobs.

Auburn joins Arkansas, Tennessee and Kentucky as SEC teams with job openings showing the huge divide in a league with six teams ranked in the Top 11.

Auburn tight end Philip Lutzenkirchen said players gave Chizik a warm ovation after the team meeting.

"I think we did what he deserved and we gave him a standing ovation and clapped for him," he said. There's so much love for coach Chizik on this team. Would we have loved to see him get another year, another opportunity? Yes, but at the same time we understand where Jay Jacobs is coming from. Three wins isn't going to cut it in our league."

Defensive tackle Jeff Whitaker said, "It's kind of crazy right now. I had a special bond with Coach Chizik."

Chizik inserted tailback Tre Mason on the final play against Alabama to get him to 1,000 yards. The next day, Mason saw his coach get fired.

"It was a rough day for everybody," Mason said. "It's tough. There are going to be a lot of tears shed because there are a lot of relationships that may be put on hold or come to an end today. I have nothing negative to say about coach Chizik. He's done a lot for me and this program. I wish him the best of luck in his future."

Chizik had sandwiched two 8-5 seasons around the national title, but never approached the success of 2010, when Newton won the Heisman Trophy. The Tigers were 7-17 in SEC games outside of 2010 during his tenure.

His hiring was criticized by some fans after Chizik went 5-19 in two seasons at Iowa State and lost the last 10 games of his first head coaching job.

Jacobs was heckled at the airport after making the hire.

Chizik had been defensive coordinator on unbeaten teams at Auburn and Texas.

A search committee comprised of former Heisman Trophy winners Pat Sullivan and Bo Jackson and former Tigers fullback Mac Crawford will assist Jacobs, the school said.

A transition year might have been expected.

Chizik had to replace the offensive and defensive coordinators after last season. Chizik made an ill-fated switch from Gus Malzahn's no-huddle, spread offense to a pro-style system with the hiring of former Temple offensive coordinator Scot Loeffler.

The Tigers struggled in the transition, partly because of shaky quarterback play and ended the season with freshman Jonathan Wallace under center.

Auburn ranked at or near the bottom of the SEC in every major statistical category offensively and defensively.

Chizik's tenure was marred by off-the-field problems, too, to the extent that he had employees of a private firm run curfew checks on players this season.

Four members of the 2010 national championship team were arrested on robbery charges in March 2011. Antonio Goodwin was convicted in April and sentenced to 15 years in prison. Dakota Mosley, Michael McNeil and Shaun Kitchens are awaiting trial.

Two-time 1,000-yard rusher Mike Dyer transferred to Arkansas State with Malzahn after being indefinitely suspended before the bowl game.

Freshman quarterback Zeke Pike was arrested in June for public intoxication and later dismissed from the team. Starting center Reese Dismukes was suspended for the opener against Clemson following a public intoxication arrest.

Auburn is also the subject of an NCAA investigation that includes the recruitment of Memphis running back Jovon Robinson, who was ruled ineligible after a guidance counselor admitted to creating a fake transcript.

The Birmingham News reported on Wednesday that NCAA investigators had interviewed at least one assistant coach and several players during the week leading up to the Iron Bowl.

Chizik's contract includes a clause that it wouldn't owe the buyout money if he is fired for cause, including findings of major rules violations or significant or repetitive violations" involving him or his program.

Chizik and Auburn have weathered past NCAA scrutiny. The governing body closed investigations into the recruitment of Newton and allegations from four former players that they were paid thousands of dollars during their college careers.

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Source: http://www.huffingtonpost.com/2012/11/25/gene-chizik-fired-auburn-football_n_2188615.html

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